Smoking is harmful to hair. Generally speaking, people seeking beauty treatment for hair loss cannot smoke. Because the nicotine in tobacco can destroy hair follicle tissue, thereby interfering with the circulation of blood and hormones in the scalp, and also increasing hormones. Moreover, smoking will cause the capillaries of the scalp to shrink, affect the normal growth function of hair, and even lead to hair loss!

It is common that smokers have rough skin, enlarged pores, and the collagen in the dermal tissue is easily lost. Naturally, the nutritional supply to the hair follicles is insufficient, and the scalp is prone to hair loss and hair breakage.

Nicotine will reduce blood oxygen concentration, hinder blood circulation in the scalp, and cause damage to hair follicles. Therefore, you should avoid smoking, maintain adequate sleep, a regular life, and keep a happy mood at all times.

Extended information:

Dangers of smoking:

1. Carcinogenesis

Smoking can reduce the activity of natural killer cells, thereby weakening the body It has the functions of monitoring, killing and clearing tumor cell growth, which further explains why smoking is a high-risk factor for the occurrence of various cancers. The incidence of laryngeal cancer in smokers is more than ten times higher than that of non-smokers, and the incidence of bladder cancer is increased by three times. This may be related to β-naphthylamine in smoke. In addition, smoking is related to the occurrence of lip cancer, tongue cancer, oral cancer, esophageal cancer, stomach cancer, colon cancer, pancreatic cancer, kidney cancer and cervical cancer.

2. Effects on the heart and cerebrovascular diseases

Smoking is the main risk factor for many cardiovascular and cerebrovascular diseases. Smokers are at risk of coronary heart disease, hypertension, cerebrovascular disease and The incidence of peripheral vascular disease increased significantly. Carbon monoxide in smoke combines with hemoglobin to form carboxyhemoglobin, which affects the oxygen-carrying capacity of red blood cells, causing tissue hypoxia, thereby inducing coronary artery spasm. Due to tissue hypoxia, compensatory polycythemia occurs and blood viscosity increases.

In addition, smoking can increase the level of plasma fibrinogen, leading to dysfunction of the coagulation system; smoking can also affect the metabolism of arachidonic acid, reducing the production of PGI2 and relatively increasing thromboxane A2, thus causing vascular dysfunction. contraction and increased platelet aggregation. All of the above may promote the occurrence and development of coronary heart disease. Due to myocardial hypoxia, myocardial stress increases and ventricular fibrillation threshold decreases. Therefore, smokers with coronary heart disease are more likely to develop arrhythmias and have an increased risk of sudden death.

3. Effects on the respiratory tract

Long-term smoking can damage and shorten the cilia of the bronchial mucosa, affecting the clearance function of the cilia. In addition, submucosal glands proliferate and enlarge, mucus secretion increases, and its composition changes, which can easily block bronchioles. In experiments on dogs, exposure to large amounts of smoke can cause emphysematous changes in the lungs.

A study by the Institute of Respiratory Diseases of China Medical University found that smokers had significantly more macrophages (AM), neutrophils (PMN) and elastase in the lower respiratory tract than non-smokers. The mechanism may be that the monocyte macrophage system in the lower respiratory tract is activated due to the stimulation of smoke particles and harmful gases. In addition to releasing elastase, the activated AM also releases PMN chemokines, allowing PMN to move from capillaries to the lungs.

Activated AM also releases macrophage growth factors to attract fibroblasts; and PMN releases large amounts of toxic oxygen free radicals and proteolytic enzymes including elastase and collagenase, which act on the lungs. of elastin, polymucin, basement membrane, and collagen fibers, leading to destruction of alveolar wall septa and interstitial fibrosis.

4. Effects on the digestive tract

Smoking can cause an increase in gastric acid secretion, generally 91.5% higher than that of non-smokers, and can inhibit the secretion of sodium bicarbonate by the pancreas, causing the duodenum to The intestinal acid load increases and induces ulcers. Nicotine in tobacco can reduce the tension of the pyloric sphincter, making it easier for bile to reflux, thereby weakening the defensive factors of the gastric and duodenal mucosa, promoting the occurrence of chronic inflammation and ulcers, and delaying the healing of original ulcers. In addition, smoking can reduce the tension of the lower esophageal sphincter and easily cause reflux esophagitis.

Baidu Encyclopedia: Smoking